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colorectal adenocarcinoma cell line hct15  (ATCC)


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    Structured Review

    ATCC colorectal adenocarcinoma cell line hct15
    Colorectal Adenocarcinoma Cell Line Hct15, supplied by ATCC, used in various techniques. Bioz Stars score: 97/100, based on 1620 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/colorectal adenocarcinoma cell line hct15/product/ATCC
    Average 97 stars, based on 1620 article reviews
    colorectal adenocarcinoma cell line hct15 - by Bioz Stars, 2026-05
    97/100 stars

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    ATCC colorectal adenocarcinoma cell line hct15
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    hct 15  (ATCC)
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    hct15  (ATCC)
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    ATCC hct15
    Hct15, supplied by ATCC, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    ATCC human colorectal carcinoma cell line hct 15
    Cytotoxic and anti-cancer activity of the N1: (A) dose-dependent inhibition of cell proliferation in murine colon carcinoma cell lines (CT26 and MC-38) and human colorectal cancer cell line <t>(HCT-15)</t> upon treatment with increasing concentrations of N1, determined by MTT assay; (B) cell viability of normal murine fibroblast cells (NIH-3T3) following treatment with N1 at indicated concentrations, demonstrating minimal cytotoxicity; (C–E) dose–response curves showing percentage growth inhibition and corresponding IC 50 values for CT26 (C), MC-38 (D), and HCT-15 (E) cells. IC 50 values are calculated by nonlinear regression analysis using GraphPad Prism. Data are expressed as mean ± SD ( n = 3).
    Human Colorectal Carcinoma Cell Line Hct 15, supplied by ATCC, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Average 97 stars, based on 1 article reviews
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    ATCC human colorectal cancer cell line hct 15
    Cytotoxic and anti-cancer activity of the N1: (A) dose-dependent inhibition of cell proliferation in murine colon carcinoma cell lines (CT26 and MC-38) and human colorectal cancer cell line <t>(HCT-15)</t> upon treatment with increasing concentrations of N1, determined by MTT assay; (B) cell viability of normal murine fibroblast cells (NIH-3T3) following treatment with N1 at indicated concentrations, demonstrating minimal cytotoxicity; (C–E) dose–response curves showing percentage growth inhibition and corresponding IC 50 values for CT26 (C), MC-38 (D), and HCT-15 (E) cells. IC 50 values are calculated by nonlinear regression analysis using GraphPad Prism. Data are expressed as mean ± SD ( n = 3).
    Human Colorectal Cancer Cell Line Hct 15, supplied by ATCC, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/human colorectal cancer cell line hct 15/product/ATCC
    Average 97 stars, based on 1 article reviews
    human colorectal cancer cell line hct 15 - by Bioz Stars, 2026-05
    97/100 stars
      Buy from Supplier

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    Cytotoxic and anti-cancer activity of the N1: (A) dose-dependent inhibition of cell proliferation in murine colon carcinoma cell lines (CT26 and MC-38) and human colorectal cancer cell line (HCT-15) upon treatment with increasing concentrations of N1, determined by MTT assay; (B) cell viability of normal murine fibroblast cells (NIH-3T3) following treatment with N1 at indicated concentrations, demonstrating minimal cytotoxicity; (C–E) dose–response curves showing percentage growth inhibition and corresponding IC 50 values for CT26 (C), MC-38 (D), and HCT-15 (E) cells. IC 50 values are calculated by nonlinear regression analysis using GraphPad Prism. Data are expressed as mean ± SD ( n = 3).

    Journal: RSC Advances

    Article Title: Exploring an azo-uracil based nickel( ii ) complex for anticancer and phosphatase like activities

    doi: 10.1039/d6ra01587e

    Figure Lengend Snippet: Cytotoxic and anti-cancer activity of the N1: (A) dose-dependent inhibition of cell proliferation in murine colon carcinoma cell lines (CT26 and MC-38) and human colorectal cancer cell line (HCT-15) upon treatment with increasing concentrations of N1, determined by MTT assay; (B) cell viability of normal murine fibroblast cells (NIH-3T3) following treatment with N1 at indicated concentrations, demonstrating minimal cytotoxicity; (C–E) dose–response curves showing percentage growth inhibition and corresponding IC 50 values for CT26 (C), MC-38 (D), and HCT-15 (E) cells. IC 50 values are calculated by nonlinear regression analysis using GraphPad Prism. Data are expressed as mean ± SD ( n = 3).

    Article Snippet: Murine colon carcinoma cell lines CT26 and MC-38, human colorectal carcinoma cell line HCT-15, and normal murine fibroblast cell line NIH-3T3 were procured from the ATCC, USA.

    Techniques: Activity Assay, Inhibition, MTT Assay

    Effect of ROS scavenging and phosphatase inhibition on N1-induced cytotoxicity: (A) percentage cytotoxicity in CT26 cells treated with N1 (0.04 and 0.08 µg mL −1 ) in the presence or absence of N -acetyl cysteine (NAC, 5 mM) and PhosSTOP phosphatase inhibitor (1×); (B) corresponding cytotoxicity in HCT-15 cells under similar treatment conditions. NAC pre-treatment significantly reduces N1-induced cytotoxicity, indicating the involvement of ROS, while PhosSTOP treatment partially attenuates cytotoxicity, suggesting a possible contribution of phosphate-related processes. Data are expressed as mean ± SD ( n = 3; *** p < 0.001, **** p < 0.0001 vs. N1-treated group).

    Journal: RSC Advances

    Article Title: Exploring an azo-uracil based nickel( ii ) complex for anticancer and phosphatase like activities

    doi: 10.1039/d6ra01587e

    Figure Lengend Snippet: Effect of ROS scavenging and phosphatase inhibition on N1-induced cytotoxicity: (A) percentage cytotoxicity in CT26 cells treated with N1 (0.04 and 0.08 µg mL −1 ) in the presence or absence of N -acetyl cysteine (NAC, 5 mM) and PhosSTOP phosphatase inhibitor (1×); (B) corresponding cytotoxicity in HCT-15 cells under similar treatment conditions. NAC pre-treatment significantly reduces N1-induced cytotoxicity, indicating the involvement of ROS, while PhosSTOP treatment partially attenuates cytotoxicity, suggesting a possible contribution of phosphate-related processes. Data are expressed as mean ± SD ( n = 3; *** p < 0.001, **** p < 0.0001 vs. N1-treated group).

    Article Snippet: Murine colon carcinoma cell lines CT26 and MC-38, human colorectal carcinoma cell line HCT-15, and normal murine fibroblast cell line NIH-3T3 were procured from the ATCC, USA.

    Techniques: Inhibition

    Cytotoxic and anti-cancer activity of the N1: (A) dose-dependent inhibition of cell proliferation in murine colon carcinoma cell lines (CT26 and MC-38) and human colorectal cancer cell line (HCT-15) upon treatment with increasing concentrations of N1, determined by MTT assay; (B) cell viability of normal murine fibroblast cells (NIH-3T3) following treatment with N1 at indicated concentrations, demonstrating minimal cytotoxicity; (C–E) dose–response curves showing percentage growth inhibition and corresponding IC 50 values for CT26 (C), MC-38 (D), and HCT-15 (E) cells. IC 50 values are calculated by nonlinear regression analysis using GraphPad Prism. Data are expressed as mean ± SD ( n = 3).

    Journal: RSC Advances

    Article Title: Exploring an azo-uracil based nickel( ii ) complex for anticancer and phosphatase like activities

    doi: 10.1039/d6ra01587e

    Figure Lengend Snippet: Cytotoxic and anti-cancer activity of the N1: (A) dose-dependent inhibition of cell proliferation in murine colon carcinoma cell lines (CT26 and MC-38) and human colorectal cancer cell line (HCT-15) upon treatment with increasing concentrations of N1, determined by MTT assay; (B) cell viability of normal murine fibroblast cells (NIH-3T3) following treatment with N1 at indicated concentrations, demonstrating minimal cytotoxicity; (C–E) dose–response curves showing percentage growth inhibition and corresponding IC 50 values for CT26 (C), MC-38 (D), and HCT-15 (E) cells. IC 50 values are calculated by nonlinear regression analysis using GraphPad Prism. Data are expressed as mean ± SD ( n = 3).

    Article Snippet: The murine colon carcinoma cell lines CT26, human colorectal cancer cell line HCT-15, and normal murine fibroblast cell line NIH-3T3 were procured from ATCC (USA).

    Techniques: Activity Assay, Inhibition, MTT Assay

    Effect of ROS scavenging and phosphatase inhibition on N1-induced cytotoxicity: (A) percentage cytotoxicity in CT26 cells treated with N1 (0.04 and 0.08 µg mL −1 ) in the presence or absence of N -acetyl cysteine (NAC, 5 mM) and PhosSTOP phosphatase inhibitor (1×); (B) corresponding cytotoxicity in HCT-15 cells under similar treatment conditions. NAC pre-treatment significantly reduces N1-induced cytotoxicity, indicating the involvement of ROS, while PhosSTOP treatment partially attenuates cytotoxicity, suggesting a possible contribution of phosphate-related processes. Data are expressed as mean ± SD ( n = 3; *** p < 0.001, **** p < 0.0001 vs. N1-treated group).

    Journal: RSC Advances

    Article Title: Exploring an azo-uracil based nickel( ii ) complex for anticancer and phosphatase like activities

    doi: 10.1039/d6ra01587e

    Figure Lengend Snippet: Effect of ROS scavenging and phosphatase inhibition on N1-induced cytotoxicity: (A) percentage cytotoxicity in CT26 cells treated with N1 (0.04 and 0.08 µg mL −1 ) in the presence or absence of N -acetyl cysteine (NAC, 5 mM) and PhosSTOP phosphatase inhibitor (1×); (B) corresponding cytotoxicity in HCT-15 cells under similar treatment conditions. NAC pre-treatment significantly reduces N1-induced cytotoxicity, indicating the involvement of ROS, while PhosSTOP treatment partially attenuates cytotoxicity, suggesting a possible contribution of phosphate-related processes. Data are expressed as mean ± SD ( n = 3; *** p < 0.001, **** p < 0.0001 vs. N1-treated group).

    Article Snippet: The murine colon carcinoma cell lines CT26, human colorectal cancer cell line HCT-15, and normal murine fibroblast cell line NIH-3T3 were procured from ATCC (USA).

    Techniques: Inhibition